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Michael BROWNLEE, Anita and Jack Saltz Professor of Diabetes Research | Cited by 71,261 | of Albert Einstein College of Medicine, NY (AECOM) | Read 248 publications | Contact Michael BROWNLEE
7 de jun. de 2012 · Among the paper’s chief authors was Dr. Michael Brownlee, Einstein Diabetes Center’s Associate Director for Biomedical Sciences and the Anita and Jack Saltz Chair in Diabetes Research.
Yao D, Brownlee M. (2010) Hyperglycemia-induced reactive oxygen species increase expression of the receptor for advanced glycation end products (RAGE) and RAGE ligands. Diabetes. 59(1):249-55.
1 de jun. de 2005 · Insulin resistance causes mitochondrial overproduction of ROS in macrovascular endothelial cells by increasing FFA flux and oxidation. From Hofmann S, Brownlee M: Biochemistry and molecular cell biology of diabetic complications: a unifying mechanism.
Michael Brownlee 1. Affiliation. 1 Departments of Medicine and Pathology, Albert Einstein College of Medicine, F-531 1300 Morris Park Ave., Bronx, NY 10461-1602, USA. brownlee@aecom.yu.edu. PMID: 15919781. DOI: 10.2337/diabetes.54.6.1615. Publication types. Lecture. Research Support, N.I.H., Extramural. Research Support, Non-U.S. Gov't.
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13 de dic. de 2001 · Abstract. Diabetes-specific microvascular disease is a leading cause of blindness, renal failure and nerve damage, and diabetes-accelerated atherosclerosis leads to increased risk of myocardial infarction, stroke and limb amputation.
